該雜志國(guó)際簡(jiǎn)稱(chēng):FRONT MOL NEUROSCI,是由出版商Frontiers Media S.A.出版的一本致力于發(fā)布醫(yī)學(xué)研究新成果的的專(zhuān)業(yè)學(xué)術(shù)期刊。該雜志以NEUROSCIENCES研究為重點(diǎn),主要發(fā)表刊登有創(chuàng)見(jiàn)的學(xué)術(shù)論文文章、行業(yè)最新科研成果,扼要報(bào)道階段性研究成果和重要研究工作的最新進(jìn)展,選載對(duì)學(xué)科發(fā)展起指導(dǎo)作用的綜述與專(zhuān)論,促進(jìn)學(xué)術(shù)發(fā)展,為廣大讀者服務(wù)。該刊是一本國(guó)際優(yōu)秀雜志,在國(guó)際上有很高的學(xué)術(shù)影響力。
《Frontiers In Molecular Neuroscience》是一本以English為主的開(kāi)放獲取國(guó)際優(yōu)秀期刊,中文名稱(chēng)分子神經(jīng)科學(xué)前沿,本刊主要出版、報(bào)道醫(yī)學(xué)-NEUROSCIENCES領(lǐng)域的研究動(dòng)態(tài)以及在該領(lǐng)域取得的各方面的經(jīng)驗(yàn)和科研成果,介紹該領(lǐng)域有關(guān)本專(zhuān)業(yè)的最新進(jìn)展,探討行業(yè)發(fā)展的思路和方法,以促進(jìn)學(xué)術(shù)信息交流,提高行業(yè)發(fā)展。該刊已被國(guó)際權(quán)威數(shù)據(jù)庫(kù)SCIE收錄,為該領(lǐng)域相關(guān)學(xué)科的發(fā)展起到了良好的推動(dòng)作用,也得到了本專(zhuān)業(yè)人員的廣泛認(rèn)可。該刊最新影響因子為3.5,最新CiteScore 指數(shù)為5.7。
本刊近期中國(guó)學(xué)者發(fā)表的論文主要有:
Brain region-specific genome-wide deoxyribonucleic acid methylation analysis in patients with Alzheimer's disease
Frontiers in Molecular Neuroscience is a first-tier electronic journal devoted to identifying key molecules, as well as their functions and interactions, that underlie the structure, design and function of the brain across all levels. The scope of our journal encompasses synaptic and cellular proteins, coding and non-coding RNA, and molecular mechanisms regulating cellular and dendritic RNA translation. In recent years, a plethora of new cellular and synaptic players have been identified from reduced systems, such as neuronal cultures, but the relevance of these molecules in terms of cellular and synaptic function and plasticity in the living brain and its circuits has not been validated. The effects of spine growth and density observed using gene products identified from in vitro work are frequently not reproduced in vivo. Our journal is particularly interested in studies on genetically engineered model organisms (C. elegans, Drosophila, mouse), in which alterations in key molecules underlying cellular and synaptic function and plasticity produce defined anatomical, physiological and behavioral changes. In the mouse, genetic alterations limited to particular neural circuits (olfactory bulb, motor cortex, cortical layers, hippocampal subfields, cerebellum), preferably regulated in time and on demand, are of special interest, as they sidestep potential compensatory developmental effects.