雜志介紹

Traffic雜志介紹

《Traffic》是一本以English為主的未開放獲取國際優(yōu)秀期刊，中文名稱交通，本刊主要出版、報道生物學-CELL BIOLOGY領域的研究動態(tài)以及在該領域取得的各方面的經驗和科研成果，介紹該領域有關本專業(yè)的最新進展，探討行業(yè)發(fā)展的思路和方法，以促進學術信息交流，提高行業(yè)發(fā)展。該刊已被國際權威數(shù)據(jù)庫SCIE收錄，為該領域相關學科的發(fā)展起到了良好的推動作用，也得到了本專業(yè)人員的廣泛認可。該刊最新影響因子為3.6，最新CiteScore 指數(shù)為8.1。

本刊近期中國學者發(fā)表的論文主要有：

• Pseudoginsenoside-F11 alleviates oligomeric β-amyloid-induced endosome-lysosome defects in microglia.

  Author: Yao XC¹, Xue X², Zhang HT¹, Zhu MM¹, Yang XW¹, Wu CF¹, Yang JY¹.

• Clustered Regularly Interspaced Short Palindromic Repeats (CRISPR)/Cas9-mediated kif15 mutations accelerate axonal outgrowth during neuronal development and regeneration in zebrafish.

  Author: Dong Z¹, Wu S¹, Zhu C¹, Wang X², Li Y¹, Chen X¹, Liu D¹, Qiang L³, Baas PW³, Liu M¹.

• Mechanisms of the anterograde trafficking of GPCRs: Regulation of AT1R transport by interacting proteins and motifs.

  Author: Zhang M^1,2, Wu G².

• Protein kinase A-mediated phosphorylation of naked cuticle homolog 2 stimulates cell-surface delivery of transforming growth factor-α for epidermal growth factor receptor transactivation.

  Author: Cao Z^1,2, Singh B^1,2,3, Li C^4,5, Markham NO^1,2, Carrington LJ¹, Franklin JL^1,2,3,6, Graves-Deal R^1,2, Kennedy EJ⁷, Goldenring JR^2,3,6,8, Coffey RJ^1,2,3,6.

英文介紹

Traffic雜志英文介紹

Traffic encourages and facilitates the publication of papers in any field relating to intracellular transport in health and disease. Traffic papers span disciplines such as developmental biology, neuroscience, innate and adaptive immunity, epithelial cell biology, intracellular pathogens and host-pathogen interactions, among others using any eukaryotic model system. Areas of particular interest include protein, nucleic acid and lipid traffic, molecular motors, intracellular pathogens, intracellular proteolysis, nuclear import and export, cytokinesis and the cell cycle, the interface between signaling and trafficking or localization, protein translocation, the cell biology of adaptive an innate immunity, organelle biogenesis, metabolism, cell polarity and organization, and organelle movement.

All aspects of the structural, molecular biology, biochemistry, genetics, morphology, intracellular signaling and relationship to hereditary or infectious diseases will be covered. Manuscripts must provide a clear conceptual or mechanistic advance. The editors will reject papers that require major changes, including addition of significant experimental data or other significant revision.

Traffic will consider manuscripts of any length, but encourages authors to limit their papers to 16 typeset pages or less.